THE END OF GERM THEORY – A MUST-WATCH SPACEBUSTERS PRESENTATION

3 thoughts on “THE END OF GERM THEORY – A MUST-WATCH SPACEBUSTERS PRESENTATION”

1. Wonderful !!

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2. To make matters worse, salicilate poisoning caused even more deaths, especially among civilians having had their Spanish flu shots:

   Abstract

   The high case-fatality rate—especially among young adults—during the 1918–1919 influenza pandemic is incompletely understood. Although late deaths showed bacterial pneumonia, early deaths exhibited extremely “wet,” sometimes hemorrhagic lungs. The hypothesis presented herein is that aspirin contributed to the incidence and severity of viral pathology, bacterial infection, and death, because physicians of the day were unaware that the regimens (8.0–31.2 g per day) produce levels associated with hyperventilation and pulmonary edema in 33% and 3% of recipients, respectively. Recently, pulmonary edema was found at autopsy in 46% of 26 salicylate-intoxicated adults. Experimentally, salicylates increase lung fluid and protein levels and impair mucociliary clearance. In 1918, the US Surgeon General, the US Navy, and the Journal of the American Medical Association recommended use of aspirin just before the October death spike. If these recommendations were followed, and if pulmonary edema occurred in 3% of persons, a significant proportion of the deaths may be attributable to aspirin.

   Article excerpts:

   A confluence of events created a “perfect storm” for widespread salicylate toxicity. The loss of Bayer’s patent on aspirin in February 1917 allowed many manufacturers into the lucrative aspirin market. Official recommendations for aspirin therapy at toxic doses were preceded by ignorance of the unusual nonlinear kinetics of salicylate (unknown until the 1960s), which predispose to accumulation and toxicity; tins and bottles that contained no warnings and few instructions; and fear of “Spanish” influenza, an illness that had been spreading like wildfire.

   More recently, influenza deaths have been attributed to salicylate. From the 1950s to the 1980s, thousands of deaths among children following influenza and other infections (eg, Reye syndrome) were unexplained until studies identified aspirin as the major contributor [14-16], and aspirin label warnings were followed by a disappearance of the condition [17]. Reye syndrome toxicity (vomiting, hyperventilation, delirium, and coma, with brain swelling and fat in the liver and proximal renal tubules) develops after ∼4 days of salicylate therapy [14] with reported mean daily doses of 25 mg/kg [18]. (Adults with salicylate toxicity present mainly with abnormal consciousness and respiratory distress [19].) Also, a recent avian influenza A-associated fatality involved Reye syndrome and aspirin use [20], and several autopsies of persons who had avian influenza revealed hemorrhagic lungs, fatty liver changes, and swollen kidneys [21] consistent with salicylate intoxication.

   Salicylates Cause Immediate Lung Toxicity and
   May Predispose to Bacterial Infection by
   Increasing Lung Fluid and Protein Levels and
   Impairing Mucociliary Clearance
   The occurrence of pulmonary edema in humans with salicylate intoxication is well documented [19, 35]. Increased pulmonary vascular bed permeability to fluid and protein, decreases in arterial pO2, and increases in postmortem extravascular lung water followed salicylate administration in sheep [46]. Salicylate also depresses the lung’s mucociliary transport system [47].

   Aspirin Advertisements in August 1918 and a
   Series of Official Recommendations for Aspirin in
   September and Early October Preceded
   the Death Spike of October 1918
   In May 1918, usual but highly contagious influenza was publicized in Spain (hence, “Spanish influenza”) [48]. In June, after 6 weeks of usual influenza in Europe, serious pulmonary lesions and deaths increased in those “admitted to the special inf luenza centres,” especially those with an “old-standing renal lesion” [60]. In July, increased mortality of young Londoners was documented [61].

   Farbenfabriken Bayer’s worldwide efforts had left few places lacking aspirin. In the United States, Bayer’s giant factory produced aspirin under “American” management. After Bayer executives were charged with violating the Trading with the Enemies Act in August 1918, advertisements encouraged confidence in aspirin [62]. The “Spanish lady” came to the United States and struck 2000 Navy men in Boston in late August. The majority recovered, but oddly, 5%-10% developed a “very severe and massive bronchopneumonia,” which, in many, lacked an accompanying leukocytosis [63]. Influenza spread.

   Official recommendations for aspirin were issued on 13 September 1918 by the US Surgeon General [64], who stated aspirin had been used in foreign countries “apparently with much success in the relief of symptoms” (p 13), on 26 September 1918 by the US Navy [29], and on 5 October 1918 by The Journal of the American Medical Association [31]. Recommendations often suggested dose regimens that predispose to toxicity as noted above.

   Homeopaths, who thought aspirin was a poison, claimed few deaths [11, 48]. Others may have suspected that aspirin was responsible. On 23 November, 1918, Horder [68] wrote in The Lancet that, for “intensely toxic cases…aspirin and all so-called febrifuge drugs must be rigidly excluded from the treatment” (p 695)

   http://cid.oxfordjournals.org/content/49/9/1405.full

   Oxford JournalsMedicine & HealthClinical Infectious DiseasesVolume 49, Issue 9Pp. 1405-1410

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3. How many elderly today are advised to ‘take an aspirin a day’ ?

   And an annual flu shot – which the medical literature shows can cause ADE (antibody-dependent enhancement). This means that those having taken the flu shots will likely experience every little “cold” as a major event, turning into acute bronchitis and pneumonia, with many of the elderly dying unnecessarily as a result.

   Add to this the one aspirin a day and you can see why drowning in your own lung fluid is today the most likely way old people die.

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